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Studies have found that autophagy levels are low in E2-induced BPH-1 cell proliferation

We examined here the plausibility of the simplest hypothesis that the change of lipid composition and its resulting membrane properties are themselves sufficient to cause cell death (Fig

Taken together, all results observed in mentioned studies indicate a new underlying mechanism of mechanical overloading to the progression of OA, that is, the GPX4-associated ferroptosis pathway (Fig

DNA damage induces non-cell-autonomous tissue inflammation through the upregulation of cytokines and chemokines, thereby hampering systemic insulin signaling

Sekita S, Yoshihira K, Natori S, Udagawa S, Muroi T, Sugiyama Y, et al